Required Reading: (pdf file is on the Program web site)
Mechanisms by which chemokines cause the increase in adhesiveness of lymphocytes to ICAM-1, involving both changes in affinity and in the topological organization of lymphocyte surface integrin:
Optional Reading:
Good general overview of chemokines and their receptors, including the newer nomenclature:
Dominant role of LFA-1 clustering in the increased avidity of LFA-1-mediated adhesion induced by PMA or through TCR signaling (referenced repeatedly in the Constantin paper above):
The chemokine BLC and its receptor, CCR5 induces the migration of B cells into lymphoid follicles, thereby being a major factor in organizing the architecture of B cell areas:
Differential chemokine receptor expression and the selective targeting of Th1 and Th2 cells to sites of inflammation:
Randolph, D.A., G.M. Huang, C.J.L. Carruthers, L.E. Bromley, and D.D. Chaplin. 1999. The role of CCR7 in T(H)1 and T(H)2 cell localization and delivery of B cell help in vivo. Science 286:2159.
Chemokines and the specific recognition of subsets of memory T cells:
Campbell, J.J., G. Haraldsen, J. Pan, J. Rottman, S. Qin, P. Ponath, D.P. Andrew, R. Warnke, N. Ruffing, N. Kassam, L. Wu, and E.C. Butcher. 1999. The chemokine receptor CCR4 in vascular recognition by cutaneous but not intestinal memory T cells. Nature 400:776.
Involvement of a recently described chemokine in promoting the entry of a specific subset of inflammatory lymphocytes into a single anatomic site, the skin: